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Oxidized low-density lipoprotein (LDL) is a potentially hazardous type of cholesterol that is produced in the body when normal LDL cholesterol is harmed by chemical interactions with complimentary radicals. Together with inflammatory actions, totally free radicals can result in hardening of the arteries (atherosclerosis). The resulting decrease in blood flow in your arteries increases your possibilities of having a heart attack or a stroke. You can produce high levels of oxidized LDL if you have extreme complimentary radical formation or simply high LDL cholesterol levels. Westend61/ Getty Images The oxidation of LDL is believed to happen when LDL cholesterol particles in your body respond with totally free radicals; unsteady molecules that are produced as an outcome of regular metabolism, an illness, or exposure to toxins.

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The oxidized LDL itself then becomes more reactive with the surrounding tissues, which can produce swelling that leads to illness and organ damage. In particular, oxidized LDL is a risk to your cardiovascular health. LDL cholesterol molecules are not all the very same size, and some are larger than others. Smaller LDL particles are more most likely to end up being oxidized, making them more harmful to your health. Some danger factors that appear to increase the levels of oxidized LDL consist of: Taking in a diet that is high in trans fats Smoking Poorly managed diabetes Metabolic syndrome, which is typically a precursor of diabetes Direct exposure to toxins through pollution and preservatives Stress When LDL ends up being oxidized, it inhabits the inner lining (endothelium) of the body's arteries, such as the carotid arteries, the coronary arteries, and arteries that provide blood to your legs and arms.

Oxidized LDL is thought to promote the advancement of atherosclerosis, which increases your risk of having a cardiovascular disease or a stroke. LDL cholesterol in the endothelial lining of the arteries promotes the accumulation of white blood cells (lymphocytes), immune cells (dendritic cells), and inflammatory cells (macrophages) in the blood vessels. Platelets, which usually help to stop bleeding by producing embolism, can stick to areas of inflammation within the arteries. When they do, they develop hardened areas inside a capillary called plaques. Over time, more macrophages, cholesterol, and other lipids begin to build up at the website (which is normally sticky) causing the plaques to grow.